Release date: 2016-06-01

New research suggests that Alzheimer's disease is triggered by the brain's attempts to fight invading microorganisms such as bacteria or viruses. Although the specific process is not yet clear, it seems that infectious pathogens cross the blood-brain barrier leading to the formation of sticky plaques associated with brain state. So far, this hypothesis has only been validated in mice, but if the same process occurs in humans, it may help scientists learn more about Alzheimer's disease and may find more effective treatments in the future.

As Anil Ananthaswamy reported in New Scientist, beta amyloid vista from peripheral lipid membranes of nerve cells has been thought to be associated with Alzheimer's disease, although researchers still have not found enough evidence to explain why this sticky spot is Accumulate, what role do they have. All they know is that if a person suffers from Alzheimer's disease, there is usually a sticky spot in his brain. Viscosity is thought to be a natural reaction of the brain after infection. During this process, the signal between the cell and the cell is blocked, and the immune response is activated to kill the damaged cells, which may lead to memory loss and confusion. These are all symptoms associated with Alzheimer's disease.

To test this hypothesis, a group of researchers from Harvard Medical School injected bacteria into the brain of mice and found plaque formation overnight. Then they used the aphids and cultured nerve cells to do the same experiment, and obtained consistent results, which proved that when the brain received the threat of pathogens, there would be plaque formation.

But they still don't know what will happen after plaque formation. Researchers say that amyloid beta may be used to surround and kill bacteria, viruses or other pathogens that cross the blood-brain barrier. If these protein plaques are not removed in time, it is likely to trigger an immune response and other protein tangles, eventually leading to Alzheimer's disease. These results suggest that the immune pathway of the innate immune system may be a potential target for the treatment of Alzheimer's disease.

The blood-brain barrier gradually deteriorates with age, which means that harmful bacteria are more likely to pass the blood-brain barrier, which explains why Alzheimer's disease is more common in the elderly.

The next step in the study is to verify whether this new hypothesis is true in the brains of people with dementia due to dementia. Neuroscientist David Holtzman commented that this was an original new discovery.

Some scientists have thought that the formation of these sticky spots seems to be the result of aging, but the original intention of these plaques is really to help us resist the invasion of pathogens, although in the end he is harmful to us.

These findings have been published in the journal Science Translational Medicine.

Source: Bio Valley

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