In the process of radish planting, there is often a phenomenon of radish dead seedlings, which will have a certain impact on the emergence of radish. So, do you know what to do with radish dead seedlings? Let's let Xiaobian introduce it to everyone.

How to do radish dead seedlings

1. The crop is inverted.

2. In the production of carrots, strengthen management, to produce fat, what elements need to be applied, and how much scientific fertilization method is needed to reduce the amount of fertilizer residues in the soil, restore soil vigor, and improve the soil environment. Resolutely oppose blind fertilization and reduce fertilizer input.

3, with the control of carrots and dead seedlings to increase the production of special effects foliar fertilizer to control the roots of carrot dead seedlings, control the spread of dead seedlings.

4. When the herbicide is used after sowing, when the three leaves are released after the emergence of the seedlings, and the upper part of the root system begins to expand, the ground and the foliage are sprayed three times, which can achieve unexpected effects.

Of course, try to reduce the number and use of herbicides, improve soil permeability, increase soil activity, and reduce soil residues of herbicides. In this way, we can better prevent the occurrence of radish dead seedlings. I hope that the Xiaobian article will help everyone.

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Vitamins & Nutritions

Vitamins:

Vitamin A: Retinol. Carotene compounds responsible for transmitting light sensation in the retina of the eye. Deficiency leads to night blindness.

Beta carotene: An antioxidant which protects cells against oxidation damage that can lead to cancer. Beta carotene is converted, as needed, to vitamin A. Food sources of beta carotene include vegetables such as carrots, sweet potatoes, spinach and other leafy green vegetables; and fruit such as cantaloupes and apricots. Excessive carotene in the diet can temporarily yellow the skin, a condition called carotenemia, commonly seen in infants fed largely mushed carrots.

Vitamin B1: Thiamin, acts as a coenzyme in body metabolism. Deficiency leads to beriberi, a disease of the heart and nervous system.

Vitamin B2: Riboflavin, essential for the reactions of coenzymes. Deficiency causes inflammation of the lining of the mouth and skin.

Vitamin B3: Niacin, an essential part of coenzymes of body metabolism. Deficiency causes inflammation of the skin, vagina, rectum and mouth, as well as mental slowing.

Vitamin B6: Pyridoxine, a cofactor for enzymes. Deficiency leads to inflammation of the skin and mouth, nausea, vomiting, dizziness , weakness and anemia.

Folate (folic acid): Folic acid is an important factor in nucleic acid synthesis (the genetic material). Folate deficiency leads to megaloblastic anemia.

Vitamin B12: An essential factor in nucleic acid synthesis (the genetic material of all cells). Deficiency leads to megaloblastic anemia, as can be seen in pernicious anemia.

Vitamin C: Ascorbic acid, important in the synthesis of collagen, the framework protein for tissues of the body. Deficiency leads to scurvy, characterized by fragile capillaries, poor wound healing, and bone deformity in children.

Vitamin D: A steroid vitamin which promotes absorption and metabolism of calcium and phosphorus. Under normal conditions of sunlight exposure, no dietary supplementation is necessary because sunlight promotes adequate vitamin D synthesis in the skin. Deficiency can lead to osteomalacia in adults and bone deformity (rickets) in children.

Vitamin E: Deficiency can lead to anemia.

Vitamin K: An essential factor in the formation of blood clotting factors. Deficiency can lead to abnormal bleeding.

Nutritions:

For the treatment of nutritional disease, any of the nutrient-related diseases and conditions that cause illness in humans. They may include deficiencies or excesses in the diet, obesity and eating disorders, and chronic diseases such as cardiovascular disease, hypertension, cancer, and diabetes mellitus. Nutritional diseases also include developmental abnormalities that can be prevented by diet, hereditary metabolic disorders that respond to dietary treatment, the interaction of foods and nutrients with drugs, food allergies and intolerances, and potential hazards in the food supply. All of these categories are described in this article. For a discussion of essential nutrients, dietary recommendations, and human nutritional needs and concerns throughout the life cycle, see nutrition, human.

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